Free CANCER'S CAUSE, CANCER'S CURE by DPM Morton Walker Page A

the DNA in a cancer cell.
    What Beljanski discovered was that the secondary structure (the hydrogen bonding between the two nucleotides that form the base pairs) can be damaged when something interferes with the hydrogen bond reforming in the replication process.
    Beljanski was able to conclude this discovery through a series of tests on the ability of the DNA cell to absorb ultraviolet light (in other words, to make a cell glow like a fluorescent light bulb). He was able to observe that sometimes the hydrogen bonds that have been unzipped for replication to occur do not zip back up. They are prevented from doing so by molecules of chemicals commonly known as carcinogens (cancer causing substances). Carcinogens disallow the hydrogen bonds to be reformed. And therein lies the problem.
    Figure 8 shows DNA destabilization:

    1. When a DNA parent molecule is closed, it cannot be acted upon by DNA polymerase to create copies of open-stranded DNA. However, once the DNA strand has split itself and the half of the ladder is open, the DNA polymerase that is always there does what it is supposed to do: it causes the open strand to replicate itself. It will continue to do this over and over until the two sides of the DNA ladder can zip themselves back up or until the open side of the ladder is somehow killed or is rendered inert.
    2. Since carcinogens are chemicals, they can lodge themselves into the sides of the DNA ladder, and this is why they prevent the hydrogen bonds in the bases, or rungs of the ladder, from reforming: they act as a counter force to the action of the hydrogen bonds. Hydrogen bonds are weak, and the carcinogen residing in the sides of the ladder literally works to pull apart, or torque open, these weak bonds.
    3. The hydrogen bonds can’t reform, so the open-ended ladder is susceptible to the DNA polymerase which is going to do the job it’s supposed to do: make copies of the open-ended rungs of the ladder. So you get an out-of-control copying of the same unzipped DNA molecule.
    4. When the DNA in a cell doesn’t zip itself back up, a cell’s preprogrammed apoptosis somehow gets dismantled. In other words, the cell doesn’t kill itself. It keeps replicating infinitely— the hallmark of a cancer cell. This is what Dr. Beljanski labeled as “destabilization of DNA,” a phenomenon which deals exclusively with the secondary structure of the DNA. Its importance cannot be understated. The disruption of the secondary structure of the DNA double helix is a central factor in the creation of cancer. This is revolutionary, and it is a discovery that is “ripe for rediscovery and reappraisal.” In fact, some twenty-five years after Mirko Beljanski first made his observation about the secondary structure and the destabilization of DNA, these phenomena are now being described by many others in the scientific community.
     
    Beljanski’s Achievements
    This point cannot be stressed strongly enough. Dr. Beljanski made a major discovery when he observed and described destabilization of DNA. First, he observed that when the separation of tightly bound strands of DNA in cellular tissue are contaminated with carcinogens, the result is that the separated strands of the double helix fail to reform in the perfect corkscrew-shaped helix. This failure causes destabilization of the DNA molecule. Second, the destabilized molecule then starts replicating itself over and over with no end, and this causes the cells that contain the destabilized DNA to start replicating themselves over and over with no end. When more carcinogens enter the area, more destabilization of more DNA molecules occur. More cells begin replicating out of control, and eventually a cancerous tumor develops. As of yet, oncologists do not appear to have adopted Beljanski’s cancer-causation finding: that destabilization of DNA is the underlying source of cancer. Consequently, the cause of cancer continues to elude them.
    Drs. Watson, Crick, and Wilkins deserved