Free CANCER'S CAUSE, CANCER'S CURE by DPM Morton Walker

national current deficit of fourteen trillion dollars. And that’s just in a healthy cell. Cancer cells divide and replicate themselves in an out-of-control fashion—far faster than a healthy cell.
     
    How DNA Recreates Itself by Replication
    Like every form of life, individual cells have a lifespan. Rather than measure lifespan as a length of time, the lifespan of a cell is measured by how many times it can replicate itself. An average number of replications of cells observed in laboratory experiments is fifty but is theorized to be at a maximum of eighty. When a cell is no longer able to replicate, apoptosis (self-induced cell death) is the result. Self-induced cell death is exceedingly important for the overall health of body tissues. Without apoptosis, every newborn organism would otherwise in reality be just one ongoing cancer with its physiological systems overrun by excessive numbers of growing cells.
    Figure 7 shows DNA replication—the recreations of itself again and again.
    There are a series of steps that it must go through:
    1. The top of the illustration shows a “parental” DNA molecule. The rungs of the parental DNA (the bases bound together with a hydrogen bond) break apart and the sides of the ladder break off (the zipper unzips itself). The parent molecule floats off and the open-sided rungs of the ladder are ready to replicate.
    2. Open, unbonded bases can now be acted upon by the DNA polymerase, the crucial enzyme that literally makes a new copy of these open, unbonded bases (the open half of the ladder). The copy the DNA polymerase makes is actually complementary to the existing half. If you look at the illustration, you will notice that the side of the ladder that’s from the parent DNA molecule has the A, the T, the C, and the G waiting for their proper mates. The DNA polymerase makes a strand that has the corresponding T, A, G, C bases ready to fuse with the existing half.
    3. The existing half of the ladder then fuses with the replicated, complimentary half because the hydrogen bonds between the bases reform. The process continues until two identical molecules of DNA have been formed. This happens twice, once for each side of the ladder that was split from the parent DNA molecule. (One zipper is now two and both are zipped up.) In a healthy cell, this process continues in each cell until the cell reaches its maximum number of times it can divide, and its preprogrammed death, apoptosis, occurs.

    This is a remarkably efficient system, and in a perfectly healthy body it runs along smoothly. However, the two strands are bound together by hydrogen bonds whose opening and closing are susceptible to internal and external influences. When the hydrogen bonds of the base pairs fail to rezip, the message that DNA delivers may be dramatically modified.
    The hydrogen bonding is disrupted by many types of molecules that come from both inside the body and from outside substances such as pollutants that the body absorbs through air, food, water, or even through the skin. These modifications lead to the replication process malfunctioning, which brings on the appearance of various serious diseases such as cancer. It is this malfunctioning that Dr. Beljanski discovered and named DNA destabilization.
     
    DNA Destabilization
    DNA interacts with many types of molecules, and these interactions may affect the fate of the cell. DNA initiates the production of other cells, so ensuring the integrity of the DNA is of prime importance in maintaining the structural integrity of cells.
    Structural integrity, however, can be compromised by damage either to the primary structure (the order and nature of the base pairs) or the secondary structure (the hydrogen bonding of the base pairs). To reiterate an important point I made above, primary structural damage is usually caused by a mutation of a base or the activation or deactivation of a base by its binding to another molecule. This is the prevailing idea of what happens to